General Information About Night Blindness
Disease Details and Frequently Asked Questions
It is a slowly progressing genetic disorder where the rod cells, which are located in the back of the eye (retina) and enable vision in the dark, lose their function due to a structural defect.
It mostly stems from genetic mutations inherited from the mother and father. Besides this, conditions like severe Vitamin A deficiency, cataracts, and glaucoma can also secondarily impair night vision.
The disease usually begins in childhood or adolescence. The first symptom is difficulty seeing at night or in dimly lit environments (like movie theaters). As the disease progresses, peripheral vision narrows, resulting in 'tunnel vision'.
The course and progression rate of the disease vary greatly from person to person. Many patients retain their central vision for many years; however, in advanced and very severe stages, the threshold of legal blindness can be reached.
During a detailed fundus examination by an ophthalmologist, characteristic black pigment deposits are seen in the retina. A definitive diagnosis is established through visual field tests and ERG (Electroretinography), which measures the electrical activity in the retina.
There is no definitive medical cure that will completely stop the disease in its genetically inherited form. Vitamin A and omega-3 supplements are given to slow the progression. However, for certain specific patients with appropriate gene mutations, modern gene therapies (like Luxturna) offer promising results.
Our health library contents are prepared for informational purposes only and with scientific data available at the time of recording. For all your questions, concerns, diagnosis, or treatment regarding your health, please consult your doctor or a healthcare institution.
